LTC Larry Sellin, 'How CV-19 Was Made
[WION] There is ongoing censorship, even in the scientific literature, to restrict publication of information contrary to the accepted narrative that COVID-19 is naturally-occurring.
What follows is not an analysis of motivations or an indictment meant to assign blame, but a history of scientific investigation that eventually led to COVID-19.
A recent news article published in the scientific journal Nature noted, that while it is important to find the origin of COVID-19 to prevent reinfection, it has been difficult pinpointing the source.
"It is quite possible we won’t find it. In fact, it would be exceptionally lucky if we land on something," said Lucy van Dorp, a geneticist from University College London.
It may indeed be impossible to identify a natural source, if COVID-19 was the product of bioengineering.
Although there are hundreds of scientific publications on coronavirus, a few relevant to the present discussion will be highlighted.
Coronavirus research did not begin with the severe acute respiratory syndrome coronavirus (SARS, SARS-CoV or SARS-CoV-1) epidemic of 2002-2004, but it was certainly accelerated by it.
Additional impetus for studying coronaviruses arose after the 2012 outbreak of Middle East respiratory syndrome (MERS or MERS-CoV).
Much of the scientific inquiry related to those two diseases has centered on a particular component of coronaviruses called the spike glycoprotein, which carries the ability for the virus to attach itself to a human cell and gain entry.
Obviously, understanding and interfering with the processes initiated by the spike glycoprotein could have prophylactic or therapeutic value.
Most of that research effort focused on the cascade of events regulated by the protein part of the spike glycoprotein, or S-protein, which has two sections, S1, primarily responsible for binding to the human cell and S2, driving fusion with the cell membrane and entry.
The S1 section contains a sequence of amino acids, the building blocks of proteins, called the receptor binding domain (RBD), which defines the coronavirus’ ability to bind to specific receptors, whether they be human or animal.
Posted by: Besoeker 2020-05-21 |